Allelopathy: A Physiological Process with Ecological by Chang-Hung Chou (auth.), Manuel J. Reigosa, Nuria Pedrol,

By Chang-Hung Chou (auth.), Manuel J. Reigosa, Nuria Pedrol, Luís González (eds.)

This ebook offers the reader appropriate information regarding real wisdom in regards to the means of allelopathy, protecting all features from the molecular to the ecological point. certain relevance is given to the physiological and ecophysiological points of allelopathy. a number of ecosystems are studied and methodological concerns are taken into consideration in numerous various chapters. The booklet has been written to be priceless either for Ph.D. scholars and for senior researchers, so the chapters comprise all invaluable details to be learn through newcomers, yet additionally they comprise loads of beneficial info and dialogue for the initiated.

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2002). This type of alkaloid is a sugar mimic. Many of these alkaloids have biological activity greater than might be expected (Seigler, 1998). Lobeline, a 48 Basic pathways for the origin of allelopathic compounds piperidine alkaloid, inhibits DNA polymerase I, reverse transcriptase activity, and protein synthesis (Wink et al. 1999). Pyrrolizidine, quinolizidine and indolizidine alkaloids Pyrrolizidine, quinolizidine and indolizidine alkaloids are of restricted distribution in plants. Pyrrolizidine alkaloids are hepatotoxic in mammals, but are involved in many 49 David S.

The most widely studied saponins for allelopathic interactions are those of alfalfa. The saponins of alfalfa (Medicago sativa) consist of at least 30 glycosides based on a variety of triterpenoid nuclei (Bialy et al. 1999, Oleszek et al. 1999). Alfalfa roots often contain as much as 4-5% saponins. These saponins consist of mono-, bi-, and tridesmosides and have from 2-7 sugar moieties attached. These saponins are responsible for many of the antinutritional aspects of alfalfa, and may also be responsible for the autotoxic effects and allelopathy attributed to this species (Oleszek et al.

1,8-Cineole inhibits all stages of mitosis. Both 1,4- and 1,8-cineole are strong growth inhibitors. The molecular target for 1,4-cineole has been shown to be asparagines synthetase. Camphor also has effects on mitosis and respiration, similar to those of 1,8-cineole (Duke and Oliva, 2004). Many volatile monoterpenes have seed-germination inhibition. These substances also inhibit growth of seedlings and roots (Cutler, 1992; Seigler, 1998). Much work involving monoterpenes has been carried out in the California chaparral community, the Florida scrub, and with species of Eucalyptus.

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